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The conclusions that can be drawn from the results is that Sulfolobus Spindle Shaped Virus (SSV) developed an escape mutation whenever a dip in the bacterial population of Sulfolobus was observed. This means that at any of these instances the SSV was able to mutate to again be able to kill the Sulfolobus. On the graph when there was more diversity and distribution within the immune systems of Sulfolobus the bacterial cells were significantly less susceptible to SSV infection. From the results it can also be concluded that Sulfolobus and SSV have a mutualistic relationship. The SSV uses host machinery to create and secrete a toxin that kills healthy uninfected cells. This also implies that infected cells are immune to the toxin is that the SSV provides infected cells with an antitoxin keeping them safe. This relationship is considered mutualism because the bacterial cells essentially want to be infected in order to survive.